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Corning Life Sciences transwell chambers corning
Transwell Chambers Corning, supplied by Corning Life Sciences, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/chambers+corning/transwell+chambers+corning/pm40613031-129-0-2
Average 90 stars, based on 1 article reviews
transwell chambers corning - by Bioz Stars, 2026-09
90/100 stars

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Related Articles

Transwell Assay:

Article Title: TRIM36 serves as a prognostic indicator linked to immune infiltration in KIRC.
Article Snippet: Migration research was aided by transwell tests conducted in particular chambers (Corning, New York, USA).

Article Title: Melatonin reverses EGFR-TKI therapeutic resistance by modulating crosstalk between circadian-related gene signature and immune infiltration patterns in patients with COVID-19 and lung adenocarcinoma.
Article Snippet: Background: Patients with lung cancer exhibit the poorest outcomes when infected with coronavirus disease 2019 (COVID-19).. However, the potential impact of COVID-19 on the tumor microenvironment (TME) of lung adenocarcinoma (LUAD) remains unknown.. Methods: Expression data and clinical information were sourced from the Cancer Genome Atlas (TCGA) and Gene Expression Omnibus (GEO) databases.

Article Title: KLF9 mediates NLRP3 inflammasome and reactive oxygen species to mediate pyroptosis in trophoblasts.
Article Snippet: The transwell assay was set up using chambers from Corning (NY, USA), featuring an 8 μm pore size, and coated with Matrigel.

Article Title: hsa_circ_0000519 promotes the progression of lung adenocarcinoma through the hsa-miR-1296-5p/DARS axis
Article Snippet: Transwell invasion and migration experiments, employing chambers (Corning, USA) covered with or without Matrigel (BD Biosciences, USA) on the upper face of the membrane, were used to assess LUAD cell invasion and migration.

Article Title: CSNK1E is involved in TGF-β1 induced epithelial mesenchymal transformationas and related to melanoma immune heterogeneity
Article Snippet: Chambers (Corning, United States) were placed in each well of a 24-well plate, and the cells were evenly seeded into the chambers at a density of 4 × 10 4 cells per well.

Article Title: A novel tertiary lymphoid structure-associated signature accurately predicts patient prognosis and facilitates the selection of personalized treatment strategies for HNSCC
Article Snippet: Cell migration and invasion assays were performed using Chambers (Corning, USA) and BioCoat Matrigel Invasion Chambers (Corning, USA) , respectively.

Article Title: Elevated expression of WSB2 degrades p53 and activates the IGFBP3-AKT-mTOR-dependent pathway to drive hepatocellular carcinoma.
Article Snippet: Chambers with an 8 μm pore size (Corning, USA) were used as previously described.

Article Title: Nucleus‐localized circSLC39A5 suppresses hepatocellular carcinoma development by binding to STAT1 to regulate TDG transcription
Article Snippet: Chambers (Corning) were preactivated with serum‐free medium in an incubator at 37°C, then 400 μL of cell suspension was placed in the top chamber, and 700 μL containing 10% FBS medium was placed in the lower chamber as an elicitor.

Pore Size:

Article Title: TRIM36 serves as a prognostic indicator linked to immune infiltration in KIRC.
Article Snippet: Migration research was aided by transwell tests conducted in particular chambers (Corning, New York, USA).

Article Title: Melatonin reverses EGFR-TKI therapeutic resistance by modulating crosstalk between circadian-related gene signature and immune infiltration patterns in patients with COVID-19 and lung adenocarcinoma.
Article Snippet: Background: Patients with lung cancer exhibit the poorest outcomes when infected with coronavirus disease 2019 (COVID-19).. However, the potential impact of COVID-19 on the tumor microenvironment (TME) of lung adenocarcinoma (LUAD) remains unknown.. Methods: Expression data and clinical information were sourced from the Cancer Genome Atlas (TCGA) and Gene Expression Omnibus (GEO) databases.

Article Title: KLF9 mediates NLRP3 inflammasome and reactive oxygen species to mediate pyroptosis in trophoblasts.
Article Snippet: The transwell assay was set up using chambers from Corning (NY, USA), featuring an 8 μm pore size, and coated with Matrigel.

Article Title: hsa_circ_0000519 promotes the progression of lung adenocarcinoma through the hsa-miR-1296-5p/DARS axis
Article Snippet: Transwell invasion and migration experiments, employing chambers (Corning, USA) covered with or without Matrigel (BD Biosciences, USA) on the upper face of the membrane, were used to assess LUAD cell invasion and migration.

Article Title: CSNK1E is involved in TGF-β1 induced epithelial mesenchymal transformationas and related to melanoma immune heterogeneity
Article Snippet: Chambers (Corning, United States) were placed in each well of a 24-well plate, and the cells were evenly seeded into the chambers at a density of 4 × 10 4 cells per well.

Article Title: A novel tertiary lymphoid structure-associated signature accurately predicts patient prognosis and facilitates the selection of personalized treatment strategies for HNSCC
Article Snippet: Cell migration and invasion assays were performed using Chambers (Corning, USA) and BioCoat Matrigel Invasion Chambers (Corning, USA) , respectively.

Article Title: Elevated expression of WSB2 degrades p53 and activates the IGFBP3-AKT-mTOR-dependent pathway to drive hepatocellular carcinoma.
Article Snippet: Chambers with an 8 μm pore size (Corning, USA) were used as previously described.

Article Title: Nucleus‐localized circSLC39A5 suppresses hepatocellular carcinoma development by binding to STAT1 to regulate TDG transcription
Article Snippet: Chambers (Corning) were preactivated with serum‐free medium in an incubator at 37°C, then 400 μL of cell suspension was placed in the top chamber, and 700 μL containing 10% FBS medium was placed in the lower chamber as an elicitor.

Migration:

Article Title: TRIM36 serves as a prognostic indicator linked to immune infiltration in KIRC.
Article Snippet: Migration research was aided by transwell tests conducted in particular chambers (Corning, New York, USA).

Article Title: Melatonin reverses EGFR-TKI therapeutic resistance by modulating crosstalk between circadian-related gene signature and immune infiltration patterns in patients with COVID-19 and lung adenocarcinoma.
Article Snippet: Background: Patients with lung cancer exhibit the poorest outcomes when infected with coronavirus disease 2019 (COVID-19).. However, the potential impact of COVID-19 on the tumor microenvironment (TME) of lung adenocarcinoma (LUAD) remains unknown.. Methods: Expression data and clinical information were sourced from the Cancer Genome Atlas (TCGA) and Gene Expression Omnibus (GEO) databases.

Article Title: KLF9 mediates NLRP3 inflammasome and reactive oxygen species to mediate pyroptosis in trophoblasts.
Article Snippet: The transwell assay was set up using chambers from Corning (NY, USA), featuring an 8 μm pore size, and coated with Matrigel.

Article Title: hsa_circ_0000519 promotes the progression of lung adenocarcinoma through the hsa-miR-1296-5p/DARS axis
Article Snippet: Transwell invasion and migration experiments, employing chambers (Corning, USA) covered with or without Matrigel (BD Biosciences, USA) on the upper face of the membrane, were used to assess LUAD cell invasion and migration.

Article Title: CSNK1E is involved in TGF-β1 induced epithelial mesenchymal transformationas and related to melanoma immune heterogeneity
Article Snippet: Chambers (Corning, United States) were placed in each well of a 24-well plate, and the cells were evenly seeded into the chambers at a density of 4 × 10 4 cells per well.

Article Title: A novel tertiary lymphoid structure-associated signature accurately predicts patient prognosis and facilitates the selection of personalized treatment strategies for HNSCC
Article Snippet: Cell migration and invasion assays were performed using Chambers (Corning, USA) and BioCoat Matrigel Invasion Chambers (Corning, USA) , respectively.

Article Title: Elevated expression of WSB2 degrades p53 and activates the IGFBP3-AKT-mTOR-dependent pathway to drive hepatocellular carcinoma.
Article Snippet: Chambers with an 8 μm pore size (Corning, USA) were used as previously described.

Article Title: Nucleus‐localized circSLC39A5 suppresses hepatocellular carcinoma development by binding to STAT1 to regulate TDG transcription
Article Snippet: Chambers (Corning) were preactivated with serum‐free medium in an incubator at 37°C, then 400 μL of cell suspension was placed in the top chamber, and 700 μL containing 10% FBS medium was placed in the lower chamber as an elicitor.

Invasion Assay:

Article Title: TRIM36 serves as a prognostic indicator linked to immune infiltration in KIRC.
Article Snippet: Migration research was aided by transwell tests conducted in particular chambers (Corning, New York, USA).

Article Title: Melatonin reverses EGFR-TKI therapeutic resistance by modulating crosstalk between circadian-related gene signature and immune infiltration patterns in patients with COVID-19 and lung adenocarcinoma.
Article Snippet: Background: Patients with lung cancer exhibit the poorest outcomes when infected with coronavirus disease 2019 (COVID-19).. However, the potential impact of COVID-19 on the tumor microenvironment (TME) of lung adenocarcinoma (LUAD) remains unknown.. Methods: Expression data and clinical information were sourced from the Cancer Genome Atlas (TCGA) and Gene Expression Omnibus (GEO) databases.

Article Title: KLF9 mediates NLRP3 inflammasome and reactive oxygen species to mediate pyroptosis in trophoblasts.
Article Snippet: The transwell assay was set up using chambers from Corning (NY, USA), featuring an 8 μm pore size, and coated with Matrigel.

Article Title: hsa_circ_0000519 promotes the progression of lung adenocarcinoma through the hsa-miR-1296-5p/DARS axis
Article Snippet: Transwell invasion and migration experiments, employing chambers (Corning, USA) covered with or without Matrigel (BD Biosciences, USA) on the upper face of the membrane, were used to assess LUAD cell invasion and migration.

Article Title: CSNK1E is involved in TGF-β1 induced epithelial mesenchymal transformationas and related to melanoma immune heterogeneity
Article Snippet: Chambers (Corning, United States) were placed in each well of a 24-well plate, and the cells were evenly seeded into the chambers at a density of 4 × 10 4 cells per well.

Article Title: A novel tertiary lymphoid structure-associated signature accurately predicts patient prognosis and facilitates the selection of personalized treatment strategies for HNSCC
Article Snippet: Cell migration and invasion assays were performed using Chambers (Corning, USA) and BioCoat Matrigel Invasion Chambers (Corning, USA) , respectively.

Article Title: Elevated expression of WSB2 degrades p53 and activates the IGFBP3-AKT-mTOR-dependent pathway to drive hepatocellular carcinoma.
Article Snippet: Chambers with an 8 μm pore size (Corning, USA) were used as previously described.

Article Title: Nucleus‐localized circSLC39A5 suppresses hepatocellular carcinoma development by binding to STAT1 to regulate TDG transcription
Article Snippet: Chambers (Corning) were preactivated with serum‐free medium in an incubator at 37°C, then 400 μL of cell suspension was placed in the top chamber, and 700 μL containing 10% FBS medium was placed in the lower chamber as an elicitor.

Membrane:

Article Title: TRIM36 serves as a prognostic indicator linked to immune infiltration in KIRC.
Article Snippet: Migration research was aided by transwell tests conducted in particular chambers (Corning, New York, USA).

Article Title: Melatonin reverses EGFR-TKI therapeutic resistance by modulating crosstalk between circadian-related gene signature and immune infiltration patterns in patients with COVID-19 and lung adenocarcinoma.
Article Snippet: Background: Patients with lung cancer exhibit the poorest outcomes when infected with coronavirus disease 2019 (COVID-19).. However, the potential impact of COVID-19 on the tumor microenvironment (TME) of lung adenocarcinoma (LUAD) remains unknown.. Methods: Expression data and clinical information were sourced from the Cancer Genome Atlas (TCGA) and Gene Expression Omnibus (GEO) databases.

Article Title: KLF9 mediates NLRP3 inflammasome and reactive oxygen species to mediate pyroptosis in trophoblasts.
Article Snippet: The transwell assay was set up using chambers from Corning (NY, USA), featuring an 8 μm pore size, and coated with Matrigel.

Article Title: hsa_circ_0000519 promotes the progression of lung adenocarcinoma through the hsa-miR-1296-5p/DARS axis
Article Snippet: Transwell invasion and migration experiments, employing chambers (Corning, USA) covered with or without Matrigel (BD Biosciences, USA) on the upper face of the membrane, were used to assess LUAD cell invasion and migration.

Article Title: CSNK1E is involved in TGF-β1 induced epithelial mesenchymal transformationas and related to melanoma immune heterogeneity
Article Snippet: Chambers (Corning, United States) were placed in each well of a 24-well plate, and the cells were evenly seeded into the chambers at a density of 4 × 10 4 cells per well.

Article Title: A novel tertiary lymphoid structure-associated signature accurately predicts patient prognosis and facilitates the selection of personalized treatment strategies for HNSCC
Article Snippet: Cell migration and invasion assays were performed using Chambers (Corning, USA) and BioCoat Matrigel Invasion Chambers (Corning, USA) , respectively.

Article Title: Elevated expression of WSB2 degrades p53 and activates the IGFBP3-AKT-mTOR-dependent pathway to drive hepatocellular carcinoma.
Article Snippet: Chambers with an 8 μm pore size (Corning, USA) were used as previously described.

Article Title: Nucleus‐localized circSLC39A5 suppresses hepatocellular carcinoma development by binding to STAT1 to regulate TDG transcription
Article Snippet: Chambers (Corning) were preactivated with serum‐free medium in an incubator at 37°C, then 400 μL of cell suspension was placed in the top chamber, and 700 μL containing 10% FBS medium was placed in the lower chamber as an elicitor.



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Functional analysis of SNRPB knockdown in HCC cells. ( A ) Validation of SNRPB knockdown efficiency in SK-HEP-1 and HCCLM3 cells at both mRNA and protein levels using two effective shRNAs (shSNRPB-2 and shSNRPB-3). ( B ) CCK-8 assay demonstrates the inhibitory effects of SNRPB knockdown on HCC cell proliferation in SK-HEP-1 and HCCLM3 cells. ( C ) Colony formation assay shows that SNRPB knockdown significantly suppresses the clonogenic capacity of HCC cells. ( D ) Flow cytometry analysis reveals that SNRPB knockdown significantly increases apoptosis rates in SK-HEP-1 and HCCLM3 cells. ( E ) <t>Transwell</t> assay demonstrates that silencing SNRPB significantly reduces the migratory capacity of HCC cells. ( F ) The pictures taken from the excised xenografts of indicated experimental groups. ( G ) Tumor volume measurements in subcutaneous xenograft tumor models showing the effects of SNRPB knockdown on tumor growth. ( H ) Tumor weight analysis at the time of sacrifice in xenograft models. ( I ) IHC staining of tumor tissues showing Ki67 expression levels. Data were drawn as mean ± SD ( n ≥ 3). * P < 0.05, ** P < 0.01, *** P < 0.001
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Functional analysis of SNRPB knockdown in HCC cells. ( A ) Validation of SNRPB knockdown efficiency in SK-HEP-1 and HCCLM3 cells at both mRNA and protein levels using two effective shRNAs (shSNRPB-2 and shSNRPB-3). ( B ) CCK-8 assay demonstrates the inhibitory effects of SNRPB knockdown on HCC cell proliferation in SK-HEP-1 and HCCLM3 cells. ( C ) Colony formation assay shows that SNRPB knockdown significantly suppresses the clonogenic capacity of HCC cells. ( D ) Flow cytometry analysis reveals that SNRPB knockdown significantly increases apoptosis rates in SK-HEP-1 and HCCLM3 cells. ( E ) <t>Transwell</t> assay demonstrates that silencing SNRPB significantly reduces the migratory capacity of HCC cells. ( F ) The pictures taken from the excised xenografts of indicated experimental groups. ( G ) Tumor volume measurements in subcutaneous xenograft tumor models showing the effects of SNRPB knockdown on tumor growth. ( H ) Tumor weight analysis at the time of sacrifice in xenograft models. ( I ) IHC staining of tumor tissues showing Ki67 expression levels. Data were drawn as mean ± SD ( n ≥ 3). * P < 0.05, ** P < 0.01, *** P < 0.001
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Functional analysis of SNRPB knockdown in HCC cells. ( A ) Validation of SNRPB knockdown efficiency in SK-HEP-1 and HCCLM3 cells at both mRNA and protein levels using two effective shRNAs (shSNRPB-2 and shSNRPB-3). ( B ) CCK-8 assay demonstrates the inhibitory effects of SNRPB knockdown on HCC cell proliferation in SK-HEP-1 and HCCLM3 cells. ( C ) Colony formation assay shows that SNRPB knockdown significantly suppresses the clonogenic capacity of HCC cells. ( D ) Flow cytometry analysis reveals that SNRPB knockdown significantly increases apoptosis rates in SK-HEP-1 and HCCLM3 cells. ( E ) <t>Transwell</t> assay demonstrates that silencing SNRPB significantly reduces the migratory capacity of HCC cells. ( F ) The pictures taken from the excised xenografts of indicated experimental groups. ( G ) Tumor volume measurements in subcutaneous xenograft tumor models showing the effects of SNRPB knockdown on tumor growth. ( H ) Tumor weight analysis at the time of sacrifice in xenograft models. ( I ) IHC staining of tumor tissues showing Ki67 expression levels. Data were drawn as mean ± SD ( n ≥ 3). * P < 0.05, ** P < 0.01, *** P < 0.001
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Functional analysis of SNRPB knockdown in HCC cells. ( A ) Validation of SNRPB knockdown efficiency in SK-HEP-1 and HCCLM3 cells at both mRNA and protein levels using two effective shRNAs (shSNRPB-2 and shSNRPB-3). ( B ) CCK-8 assay demonstrates the inhibitory effects of SNRPB knockdown on HCC cell proliferation in SK-HEP-1 and HCCLM3 cells. ( C ) Colony formation assay shows that SNRPB knockdown significantly suppresses the clonogenic capacity of HCC cells. ( D ) Flow cytometry analysis reveals that SNRPB knockdown significantly increases apoptosis rates in SK-HEP-1 and HCCLM3 cells. ( E ) Transwell assay demonstrates that silencing SNRPB significantly reduces the migratory capacity of HCC cells. ( F ) The pictures taken from the excised xenografts of indicated experimental groups. ( G ) Tumor volume measurements in subcutaneous xenograft tumor models showing the effects of SNRPB knockdown on tumor growth. ( H ) Tumor weight analysis at the time of sacrifice in xenograft models. ( I ) IHC staining of tumor tissues showing Ki67 expression levels. Data were drawn as mean ± SD ( n ≥ 3). * P < 0.05, ** P < 0.01, *** P < 0.001

Journal: Journal of Experimental & Clinical Cancer Research : CR

Article Title: SNRPB/CCNB1 axis promotes hepatocellular carcinoma progression and cisplatin resistance through enhancing lipid metabolism reprogramming

doi: 10.1186/s13046-025-03463-y

Figure Lengend Snippet: Functional analysis of SNRPB knockdown in HCC cells. ( A ) Validation of SNRPB knockdown efficiency in SK-HEP-1 and HCCLM3 cells at both mRNA and protein levels using two effective shRNAs (shSNRPB-2 and shSNRPB-3). ( B ) CCK-8 assay demonstrates the inhibitory effects of SNRPB knockdown on HCC cell proliferation in SK-HEP-1 and HCCLM3 cells. ( C ) Colony formation assay shows that SNRPB knockdown significantly suppresses the clonogenic capacity of HCC cells. ( D ) Flow cytometry analysis reveals that SNRPB knockdown significantly increases apoptosis rates in SK-HEP-1 and HCCLM3 cells. ( E ) Transwell assay demonstrates that silencing SNRPB significantly reduces the migratory capacity of HCC cells. ( F ) The pictures taken from the excised xenografts of indicated experimental groups. ( G ) Tumor volume measurements in subcutaneous xenograft tumor models showing the effects of SNRPB knockdown on tumor growth. ( H ) Tumor weight analysis at the time of sacrifice in xenograft models. ( I ) IHC staining of tumor tissues showing Ki67 expression levels. Data were drawn as mean ± SD ( n ≥ 3). * P < 0.05, ** P < 0.01, *** P < 0.001

Article Snippet: The assessment of cell migration was conducted using a Transwell chamber (Corning).

Techniques: Functional Assay, Knockdown, Biomarker Discovery, CCK-8 Assay, Colony Assay, Flow Cytometry, Transwell Assay, Immunohistochemistry, Expressing